Friday, March 13, 2009
Who am I?
Thursday, March 12, 2009
Hannah's Diary
Sunday, March 01, 2009
Heart Disease- Part Two - Insulin Resistance

Last time I talked about the dangers of body fat, a belly that sticks out, becoming a diabetic, and diabetes in general. I thought I'd try to attempt to explain why this is the case. The above slide explains it on a very simple level, I'm guessing most of you have no idea what it means. HOWEVER, after reading this I'm hoping you well be able to understand this slide, and understand the dangers of insulin resistance. Maybe you won't be able to read it, but you'll know the story behind the slide.
Let me start by saying insulin resistance leads to type 2 diabetes. Many of you who have a belly, elevated blood pressure, and not a regular exerciser (Walk and hour a day and you're and exerciser) most likely have insulin resistance going on. You're headed toward type 2 diabetes that leads to so many other wonderful problems.
So what is insulin resistance? Let start by explaining normal metabolism. After you eat, in a response to your fed state, beta cells in your pancreas release insulin. This signals tissues that use glucose in your body (Fat tissue & Muscle tissue) to absorb glucose. Simple enough right? You eat, and your body recognizes you ate by the increase of glucose in your blood stream, it releases insulin (which is used to move glucose into muscle and fat tissue), and then your blood glucose levels drop to normal again.
In a person who is insulin resistant it requires more insulin than the normal person to get glucose into the muscle and fat tissue after you eat. Let's talk about why that might be.
We in our culture live in a constantly fed state. Our fat and muscle cells are constantly being forced to accept more than they have ability to accept. Part of this problem as well is that every time these cells touch insulin they get worn down a little, and it takes a little more insulin to get the job done. Remember that point last point. We'll talk about how to fix that later. Eventually it requires our pancreas to produce a lot more insulin than normal to get the job done after every time we eat because our tissues are constantly being hammered with more food/glucose than it can handle. This in turn leaves us with a higher blood glucose levels a lot of the time.
Side note- Remember, every time your get your blood labs done you're required to fast. Well this fasting can hide insulin resistance until BAM! Things are way out of control, and now your find out you're a type 2 diabetic. The problem is that it's difficult to get accurate readings on blood labs if you're not fasting.
So let's take the next step. Not only does fat tissue try to stop accepting glucose it also releases free fatty acid into the blood stream which is converted by the liver into triglycerides. So now we have an elevated trig level problem. Well I'm telling you right now it's a big, big problem. This increase in Trigs ruins, wrecks, and breaks normal lipid metabolism problems. Simply stated, it creates a horrible cholesterol problem that MOST doctors and many cardiologist don't take seriously. The reason why is because it's not just an LDL problem. LDL is commonly called "bad cholesterol." The problem is, that there is so much more to the story than just bad cholesterol.
So here is the next step, and I can't explain it completely and keep it simple, so I'm just going to keep it simple and leave out some detail. This increase in triglycerides does two things. One it effects your good cholesterol, and two it changes your bad cholesterol.
In regards to good cholesterol:
- One it lowers the total number of good cholesterol levels in your blood. That is bad, bad, bad, and many doctors won't treat this problem until it's way out of control. Even then, many doctors won't touch it at all because they aren't convinced it's a problem. TAKE MY WORD FOR IT! Low levels of good cholesterol is a very bad problem.
- It make the good cholesterol that you have less effective. That is bad new my friends. The good cholesterol is what keeps your arteries from building up plaque that narrows and hardens your arteries. It has been clearly demonstrated that even if you have very, very, low levels of bad cholesterol, if you have low good cholesterol your are at great risk of a heart attack or stroke.
So what is the other part of the problem of high triglyceride problem in regard to bad cholesterol:
- It makes your LDL "bad cholesterol" even more dangerous by shrinking them in size(smaller and more dense), and increasing the total amount of bad cholesterol particles in your blood stream. This is a big problem. I'm willing to bet none of you have ever had blood test that looks for this problem. The normal blood test that you have done by your doctor does not look for this problem at all. It looks at the total weight of cholesterol being carried by your bad cholesterol in your blood, and not the total number of bad particles. That's tough to explain without a slide and I can't find one.
However, even if you have never had a blood test that looks for small dense LDL particles, you can guess you have them by these signs. You have low HDL, and elevated triglycerides. If your have type 2 diabetes you have this problem, and if you have metabolic syndrome (
Here is another kicker, this elevation of fat floating around in your blood forces your body to eventually store it as fat someplace. Where? In the belly area. Yes, it is stored all over, but predominately in the belly area for some reason. We'll talk more about why this is a problem in another post.
So where the heck is the good news in all of this? This is completely reversible! How? Activity! Movement changes those muscle fibers and makes them hungry again. They stop being insulin resistant. THAT IS THE SECRET! You have to move amigos y Amiga's.
Now listen here, you may think you can trick your body. There are diets, like Atkins, that keep your insulin levels low. Therefore your body looks to your fats stores for those Trigs that they need for fuel, and bingo you start losing weight. However, in my little mind there is a problem with this. We all know people who have done these low insulin diets (self included, I generally live a South Beach diet), they lost the weight, then go off the diet and bam they gain 50 pounds in a month. I wondered why, and in my mind it is because they never changed their insulin resistant muscles tissue through exercise. So, when they went back to a "regular diet", with no activity, their muscle which never were fixed through exercise. So they continue to resist this increase of glucose, there is a Trig problem, and bam it's all stored as fat. The crazy part to me is how quickly it can happen if you're not exercising. If you're not moving, you leave the Atkins diet and eat some pizza, heck yes it was stored as fat. Yes that gain in weight on the scale is for real. If you're running, it doesn't happen. I know this from personal experience.
So as I read this I wonder how clear I made it. If there is a couple of key points I want you to remember it is this:
- Constant eating of foods that are carb loaded eventually beats down your body, and your pancreas can't keep up with the demand of insulin over the course of years and then your pancreas shuts down.
- Your doctor isn't going to tell you that your insulin resistant until you're a diabetic. A few will, but by far the majority won't.
- This whole problem is completely reversible!
- One of the ways to absolutely reverse this problem, and the best method by far is diet and exercise.
The moral to the story for me is this, you must start moving. That belly needs to be eliminated. Don't think for a second that just because you're thin you don't have to exercise. I'll make that case in a later post. Plenty of thin people die of heart disease.
Get your ipod and go for a one hour walk every day.
A Big Day for Sam!
Monday, February 23, 2009
Heart Disease- Part One
Sunday, February 15, 2009
99 & 1
Saturday, January 31, 2009
Fasting...Mmmm, makes me hungry.
So busy!
Sunday, January 11, 2009
The Young Men from Lodi 2!
Georgia Art Work
Heaven is....
Sitting around on Saturday afternoon watching college football, and strumming the guitar.
Scary
I don't know, kind of creeped my out. This photo was taken way after bed time. I heard some noise upstairs, and look what was found.
Wednesday, January 07, 2009
"Life changing beans"
cannellini beans
Sunday, January 04, 2009
I thought you might find this interesting.
December 16, 2008
Toronto, ON - Eating a diet rich in low-glycemic foods, such pumpernickel and flaxseed breads, beans, peas, lentils, and nuts, provides greater improvements in glycemic control in diabetic patients compared with a diet rich in cereal fibers, a new study has shown. After six months of eating foods with a low-glycemic index, patients lowered glycosylated hemoglobin A1c (HbA1c) levels and raised HDL-cholesterol levels significantly more than those eating the high-cereal-fiber diet, report researchers.
"There's nothing fancy here," lead investigator Dr David Jenkins (University of Toronto, ON) told heartwire. "Slowly released carbohydrates seem to have a benefit in terms of metabolism in these diabetic patients. If you release carbohydrates into the digestive track slowly, it gets absorbed slowly, and over time the tissues become insulinized, taking up the glucose without much of it appearing in the blood or as a rise in blood sugar."
The study is published in the December 17, 2008 issue of the Journal of the American Medical Association.A nondrug option for patients
In an interview with heartwire, Jenkins said that diabetic patients are typically advised to follow a similar low-fat diet that patients with heart disease follow. Although it is recommended they reduce calories from carbohydrates, many diabetic patients have been influenced by the Atkins diet, reducing their calories from carbohydrates from the recommended 50% to approximately 40%, said Jenkins.
In this study, the researchers studied the effects of a low-glycemic-index diet on diabetes control and cardiovascular risk factors in 210 individuals with diabetes treated with antihyperglycemic medications. Previous studies have suggested the dietary strategy could improve diabetes control, as well as improve various cardiovascular risk factors, such as raising HDL cholesterol and lowering triglyceride and CRP levels.
Patients randomized to the low-glycemic-index diet consumed pumpernickel, rye, and flaxseed breads, large-flake oatmeal or oat-bran cereal, fruits such as apples and pears, pasta, beans, peas, lentils, and nuts. Among those randomized to the high-cereal-fiber diet, participants consumed whole-grain breads and breakfast cereals, brown rice, potatoes with skins, and fruits such as cantaloupe and mango, as well as crackers. Caloric intake did not differ between the two treatment arms.
After six months, HbA1c levels were significantly better in individuals assigned to the low-glycemic-index diet compared with those who consumed the high-cereal-fiber diet. HDL-cholesterol levels were also significantly improved. LDL-cholesterol and triglyceride levels were not significantly altered with the dietary strategy.
Jenkins pointed out that a diet based on a low glycemic index differs from a low-glycemic diet. The Atkins diet, an example of a low-glycemic diet, involves the restriction of carbohydrates. A low-glycemic-index diet, on the other hand, does not restrict the consumption of carbohydrates but rather restricts foods that overwhelm the insulin secretion mechanisms and cause spikes in blood glucose levels.
The reduction in HbA1c was modest, said Jenkins, but the diet "is another tool in the toolkit for clinicians" and should help those not wishing to prescribe too many antihyperglycemic medications or who do not wish to significantly increase the dose of existing drugs. Investigators did not observe any appreciable hypoglycemic occurrences, something that is difficult to avoid with medications, said Jenkins.
Saturday, January 03, 2009
Wednesday, December 31, 2008
Tuesday, December 30, 2008
Scout Outing
Our scout master arranged to have the scout visit the news studio of KCRA. They had a chance to watch a broadcast, get a tour of the studio, and sit and talk about the profession of a metorologist with Dirk Verdorn. We had a great time. Stopped by Leatherby's on the way home and ate an obscence amount of ice cream.
Sunday, December 28, 2008
Air Temp of 48F
I believe it started with "Uncle Sean, I brought my swim trunks, can I jump off your roof?"
"Kurt, it's freezing outside and it's raining..... You must have asked your Dad this question what did he say?" Kurt "He said it's fine." Me "What? Really?" Gerry "I don't care, I'm tired of trying to talk him out of doing it." Me (I kind of sound like a Mom here) "You understand if you get hurt, or the water is so cold you inhale underwater, your Dad and I aren't coming in after you right? And if you hurt yourself, you have to wait until after Christmas eve dinner to walk yourself to the ER, because you're not goofing up dinner right?" Kurt "Yeah, sure." Me "Well great, let me go get my camera then!"
Saturday, December 27, 2008
The Christmas Card That Wasn't
Suzi was kind enough to agree, so we did. However, I still like this photo alot.
Friday, December 26, 2008
It's just not right! Where is the loyalty?
Sunday, December 21, 2008
Ugh! I give up....
Shooting a Christmas card photo in our family is quite an event. A couple of words come to mind to summarise the event: madness, tears, side splitting laughter, anger, persistence, threats, compromise, and good enough.
Friday, December 19, 2008
What a girl wants!
Hannah-Oh, I'd like a starbucks card for hot chocolate.
Mom-Thats it?
Hannah- Yep.
You got what!?
Wednesday, December 17, 2008
Patrick & Sam?
Friday, December 12, 2008
Armstrong & Getty
Ding Dong!
Thursday, December 11, 2008
Tuesday, December 09, 2008
Sunday, December 07, 2008
Lodi Christmas Tree Run
3 out of the 6 Kelly's did the 5k Christmas run. We were all over NorCal as a family that day (Suzi and Sam= soccer, Hannah @ a friend's house). This was Georgia's first big run. She was the only 7 year old to run it. Most of the kids ran the "one miler", she's tough!!! The next oldest to run it was a teenager. Wow! If you ask her she'll tell you her legs are sore, and that she didn't have any fun at all. But when you remind her of the early morning doughnut, the run with the friends, all the people cheering for her, and the Thai food afterward, and then you ask her if she'd do it again, then she might say "mmmm yeah, maybe. I had a great time." Patrick, and I have done two runs together and I look forward to getting my butt kicked regularly by the young whipper snapper. Families represented in the bottom photo: Hazelhofer, Robison, Kelly, Brink, Judd, Gonzalez, and La Viola. Great times!
Monday, December 01, 2008
New Tool
Run To Feed The Hungry


Saturday, November 29, 2008
Reindeer Food
The brothers on the other hand are pains in the rear.
Patrick - "Georgia, all that's really going to do is attract raccoons to the yard."
Sam - "Yeah, Georgia. We are going to over run by raccoons Christmas eve, great."
Me - "You know you two, when you were little, no one gave you crap. No one gave you any crap for going to barney concert and loving it Patrick. So shut it you two."
Georgia - "Yeah, shut it."
It was all pretty funny. I laughed through the whole thing.
Monday, November 24, 2008
Triglycerides
Triglycerides in plasma are derived from fats eaten in foods or made in the body from other energy sources like carbohydrates. Calories ingested in a meal and not used immediately by tissues are converted to triglycerides and transported to fat cells to be stored. Hormones regulate the release of triglycerides from fat tissue so they meet the body's needs for energy between meals.